By Jeffrey L Cummings, Serge Gauthier
Given the advance of latest healing options and items, Serge Gauthier and Jeffrey Cummings have compiled an inventory of up to date subject matters that allows you to be of curiosity essentially to neurologists targeting Alzheimer's affliction, and likewise to psychiatrists and geriatricians. The members, all revered of their subspecialties, have written updated, comprehensively referenced chapters which should supply information in addition to stimulate dialogue on the place present remedy is heading.
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Extra info for Alzheimer's disease and related disorders annual, 2002
Anderson AJ, Su JH, Cotman CW. DNA damage and apoptosis in Alzheimer’s disease: colocalization with c-Jun immunoreactivity, relationship to brain area, and effect of postmortem delay. J Neurosci 1996; 16:1710–1719. 45. Troy CM, Rabacchi SA, Friedman WJ et al. Caspase-2 mediates neuronal cell death induced by beta-amyloid. J Neurosci 2000; 20:1386–1392. 46. Pike CJ, Balazs R, Cotman CW. Attenuation of beta-amyloid neurotoxicity in vitro by potassium-induced depolarization. J Neurochem 1996; 67:1774–1777.
Loo DT, Copani A, Pike CJ et al. Apoptosis is induced by beta-amyloid in cultured central nervous system neurons. Proc Natl Acad Sci USA 1993; 90:7951–7955. 44. Anderson AJ, Su JH, Cotman CW. DNA damage and apoptosis in Alzheimer’s disease: colocalization with c-Jun immunoreactivity, relationship to brain area, and effect of postmortem delay. J Neurosci 1996; 16:1710–1719. 45. Troy CM, Rabacchi SA, Friedman WJ et al. Caspase-2 mediates neuronal cell death induced by beta-amyloid. J Neurosci 2000; 20:1386–1392.
1–3 It is not clear, however, that apoptosis in AD serves to do more than remove cells that have become useless or too damaged to go on. Alternatively, programmed cell death (PCD, apoptosis) in AD could be the ultimate step in a linked series of events that starts with the earliest stresses on the neuron and progresses over time to its demise. Much of our knowledge of apoptosis is based on studies of the immune system where apoptotic events occur on a rapid time scale. In the brain, it is probable that the events leading to neuronal apoptosis are spread over a longer time, perhaps spanning years.