By Cedric S. Raine (auth.), Pierre Morell (eds.)
My colleagues and i've been gratified through how speedily the 1st variation of Myelin has been aeeepted as a customary referenee paintings through myelin researehers. this can be unquestionably the first faetor aecounting for the excessive price of reeidivism one of the authors with respeet to coaching of a seeond variation; 11 of the unique twelve participants have been back concerned. 4 new authors (Wendy Cammer, Marjorie Lees, Ute Traugott, and Seymour Greenfield) have additionally eontributed to the current quantity. This new version keeps many points of the layout of the unique, even together with use of a similar bankruptcy headings. therefore, reviews within the preface to the 1st variation in regards to the point at which fabric is gifted and the relationships among chapters overlaying the "basic" and "c1inical" fabric are nonetheless acceptable. regardless of sure similarities in total association, comparability of the current variation with the unique demonstrates that our eHorts to restriet growth within the size of the textual content weren't particularly winning; the recent version is mueh lengthier than the unique. however, the rise in textual content size is understated relative to the rise in details within the seven years setting apart the instruction of the 2 versions. many of the fabric within the person chapters has needed to be thoroughly redone with a view to comprise this new info. for instance, our view of the metabolism of myelin more and more needs to take consciousness of a truly quickly turning over pool of convinced of its components.
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ACKNOWLEDGMENTS. The collaboration and constructive criticism of Drs. Robert D. Terry, Murray B. Bornstein, William T. Norton, John W. Prineas, Kinuko Suzuki, and Ute Traugott deserve special mention. The excellent backing provided by my technical staff, Everett Swanson, Miriam Pakingan, and Howard Finch, has been an essential component of this work. I thank Ms. Mary Palumbo far her faithful secretarial assistance. This work was supported in part by USPHS Grants NS-08952 and NS-11920, by a grant from the Alfred P.
Also described at CNS nodes are desmosomes between adjacent lateralloops (Metuzals, 1964) (see Fig. , 1976) (see Figs. 28, 29, and 33), paranodal constriction of the axon with a prominent bulge in the nodal region (see Figs. 28 and 29), and synapses abutting the axolemma at the node (Andres, 1965; Bodian and Taylor, FIG URE 28. Immature CNS node of Ranvier from a 2-day-old kitten spinal cord. This shows beginning alignment of lateral loops adjacent to anode (right). There are abundant cytoplasm and many microtubules within each loop.
CENTRAL NERVOUS SYSTEM REMYELINATION It was believed for many years that in the CNS, loss of myelin represented an inexorable lesion. However, the in vitra studies of Bornstein and Appel (1961), later confirmed at the EM level (Raine and Bornstein, 1970), and the barbotage lesions of M. B. Bunge et al. (1961) proved that remyelination in the CNS was possible. Subsequently, many CNS demyclinating MORPHOLOGY OF MYELIN AND MYELINATION 37 diseases and experimental models have been shown to display remyelination (see, among others, R.