By David BenEzra (auth.), David BenEzra M.D., Ph.D.,, Stephen J. Ryan M.D., Bert M. Glaser M.D., Robert P. Murphy M.D. (eds.)
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Brit. J. Ophthalmol. , A. Debbia, F.. Guerrieri, and R. Bolzani: Abnormal blood viscosity and filtrability in retinal vein occlusion. Clin. Hemorheol. 4: 555-561 (1984) 29 7. C. D. Sanders, and G. Wetherley-Mein: Viscosity and retinal vein thrombosis. Brit. J. Ophthalmol. 60: 397-410 (1976) 8. H. S. Gragoudas: Prognosis for rubeosis iridis following central retinal vein occlusion. Brit. J. Ophthalmol. 63: 735-743 (1979) 9. O. M. : Abnormal blood viscosity and haemostasis in longstanding retinal vein occlusion.
The flow in the ophthalmic artery remained unaffected and the fluorescein concentration in that artery gradually increased in the course of about 2 s to a level at which maximal fluorescence occurs. ] After a time interval of about 2 s the lOP elevation was released, and the cine camera was started. 9 ms after lOP release). [By that time the eye movements, caused by this release. 8 s. 8 s; then the next following R-wave of the ECG triggered the release of the lOP elevation. By means of this timing procedure the fluorescein inflow into the retinal arterioles occurred in different recordings in approximately the same phase of the cardiac cycle, which improved the reproducibility, especially with regard to the measurements of flow velocities in the retinal arterioles.
RESULTS Average values of trans retinal P0 far from the large retinal vessels 2 (intervascular P0 ). 2 ~ gradients in normoxia : In normoxia intervascular P~ at 50 urn from the retinal surface was 26 mmHg based on 7 measurements. Tfie local P0 pro2 gressively decreases when the microelectrode was advanced through the inner retinal layers. The value in the outer plexiform layer (which corresponds to 60 %retinal depth) was 13 mmHg. There was a continuous increase of P0 to 2 the outer segment of the photoreceptors, where the local P0 was 55 mmHg 2 (Fig.