By Sandra Gordon-Salant, Robert D. Frisina, Richard R. Fay, Arthur N. Popper
This quantity is to compile famous scientists who research presbycusis from the point of view of complementary disciplines, for a overview of the present kingdom of data at the getting older auditory method. Age-related listening to loss (ARHL) is among the best 3 most typical power medical conditions affecting members elderly sixty five years and older. The excessive occurrence of age-related listening to loss compels audiologists, otolaryngologists, and auditory neuroscientists alike to appreciate the neural, genetic and molecular mechanisms underlying this illness. A accomplished figuring out of those elements is required in order that potent prevention, intervention, and rehabilitative concepts might be constructed to ameliorate the myriad of behavioral manifestations.
The objective is to supply scholars and researchers in auditory technological know-how and getting older with a figuring out of a few of the results of getting older at the auditory approach.
- Introduction and evaluation Sandra Gordon-Salant and Robert D. Frisina
- The body structure of Cochlear Presbyacusis Richard A. Schmiedt
- The mobile Biology and body structure of the getting older crucial Auditory Pathway Barbara Canlon, Robert Benjamin Illing, and Joseph Walton
- Closing the distance among Neurobiology and Human Presbycusis: Behavioral and Evoked power experiences of Age-related listening to Loss in Animal versions and in people James R. Ison, Kelly L. Tremblay, and Paul D. Allen
- Behavioral reviews with getting older people: listening to Sensitivity and Psychoacoustics Peter J. Fitzgibbons and Sandra Gordon-Salant.
- Binaural Processing and Auditory Asymmetries David A. Eddins and Joseph W. corridor III
- The results of Senescent adjustments in Audition and Cognition on Spoken Language Comprehension Bruce A. Schneider, Kathy Pichora-Fuller, and Meredyth Daneman
- Factors Affecting Speech realizing in Older Adults Larry E. Humes and Judy R. Dubno
- Epidemiology of Age-related listening to Impairment Karen J. Cruickshanks, Weihai Zhan, and Wenjun Zhong
- Interventions and destiny cures: classes from Animal types James F. Willott and Jochen Schacht
Sandra Gordon-Salant is Professor and Director of the Doctoral application in scientific Audiology within the division of listening to and Speech Sciences on the collage of Maryland, university Park. Robert D. Frisina is Professor of Otolaryngology, Neurobiology & Anatomy, and Biomedical Engineering, and affiliate Chair of Otolaryngology on the collage of Rochester scientific college. Arthur N. Popper is Professor within the division of Biology and Co-Director of the guts for Comparative and Evolutionary Biology of listening to on the collage of Maryland, university Park. Richard R. Fay is Director of the Parmly listening to Institute and Professor of Psychology at Loyola collage of Chicago.
About the series:
The Springer guide of Auditory learn offers a chain of man-made reports of basic issues facing auditory structures. every one quantity is self sufficient and authoritative; taken as a suite, this sequence is the definitive source within the box.
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Extra resources for The Aging Auditory System
J Gerontol 37:275–282. Hequembourg S, Liberman MC (2001) Spiral ligament pathology: a major aspect of age-related cochlear degeneration in C57BL/6 mice. J Assoc Res Otolaryngol 2:118–129. Ichimiya I, Suzuki M, Mogi G (2000) Age-related changes in the murine cochlear lateral wall. Hear Res 139:116–122. Jerger J, Chmiel R, Stach B, Spretjnak M (1993) Gender affects audiometric shape in presbyacusis. J Am Acad Audiol 4:42–49. Johnson KR, Erway LC, Cook SA, Willott JF, Zheng QY (1997) A major gene affecting age-related hearing loss in C57BL/6J mice.
2 kHz. If the hypothesis of the EP-controlled OHC amplifier gain is correct, this slope and breakpoint represent the distribution of the OHC amplifier gain along the cochlear spiral. This gain distribution may be expected to differ among species depending on the frequencydistance map of the particular cochlea. Fitting the mean furosemide gerbil data to that of the human audiometric profile is shown in the bottom panel of Fig. 7. The human data are from two sources and have been screened for noise history to minimize the effects of OHC loss.
C) Quantitative analysis in the PVCN, showing a significant increase in the total number of calbindin-immunopositive neurons. (d) Percentage of calbindin-positive neurons in the PVCN did not show any significant regression with increasing age. (From Idrizbegovic et al. 2001) differs from old rats where GFAP immunoreactivity is homogeneously distributed in all parts of the nucleus (Jalenques et al. 1995). There was no change in the total number of GFAP neurons between these two ages in any part of the cochlear nucleus except for its anteroventral subdivision, where a decrease in neuronal number occurs with age.