By Hans Hoff, Kurt Jellinger (auth.), Igor Klatzo, Franz Seitelberger (eds.)

The Symposium on mind Edema has been prepared through the Osterreichische Arbeitsgemeinschaft fur Neuropathologie and through the matter fee for Neuropathology of the area Federa­ tion of Neurology. we wish to precise our due to Dr. L. van Bogaert, president of the W.F.N., for his nice curiosity within the layout of this convention and for his full of life help. one of many goals ofthe challenge fee for Neuropathology has been to give a contribution to the advances in a variety of difficulties that are changing into ripe for elucidation by means of specialists in several fields. the belief of the Symposium on mind Edema has as its history a compelling necessity to clarify this topic that is clinically so vital and neuropathologic best friend so poorly understood. It was once was hoping that this is able to be accomplished by way of a multidisciplinary strategy related to various features to be mentioned and correlated. mind edema, being an irregular cerebral situation, belongs essentially to the sector of neuropathology, yet to achieve success in our endeavors we needed to observe neuropathology in its latest and widest phrases. by means of this we remember the fact that such neuropathology comprises the examine of all irregular phenomena within the anxious tissue concerning the structural adjustments, and via "structural" we suggest not just tissual or mobile ones but in addition alterations on ultrastructural and molecular degrees. therefore we would have liked shut and built-in coopera­ tion of specialists in fields equivalent to neurohistology, histochemistry, electron microscopy, neurochemistry, and neurophysiology.

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Extra resources for Brain Edema: Proceedings of the Symposium September 11–13, 1965, Vienna

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202, 155-176 (1961). _ _ and F. W. Kolkmann: Zur Ultrastruktur des perifokalen und histotoxischen Hirn5dems bei der Ratte, Acta Neuropath. (Berlin), 1, 519-526 (1962). Walker, E. A. and J. J. Adamkiewicz: Pseudotumor cerebri associated with prolonged corticosteroid therapy, J. Amer. Med. Assn. 188, 779-784 (1964). A. EARL WALKER BRAIN EDEMA AS A NEUROLOGICAL PROBLEM-II Brain edema resulting from local involvement as the result of trauma, surgical procedures, vascular accidents, tumors or inflammation of the brain has been recognized for some time.

0 ,p 0 ::1. o NORMAL o 24 48 Hrs. after deflation of ballo on Fig. 11. The reduction in ganglioside content is maximal after 24 hours of compression. The depression in cerebral activity follows a roughly comparable course. the change is roughly proportional to the deterioration of the animal's condition. There is no obvious change in cerebroside content in either the white matter or in the brain stem. Gangliosides are known to be related not only to the excitability of the cerebral tissue, but they are also involved in the energyassisted ion movement across the cell membrane ru, 32, 33, 34).

These three (or four) elements are contained in a "closed box," subdivided by partitions, the falx (f) and tentorium (t) within which subdivisions mechanical shifts of the cerebral elements may occur. " Herniations may occur under the falx, through the tentorial opening and through the foramen magnum. elucidate some of the problems with which the clinical neurosurgeon has to deal (1,],~, 10,11). " The present situation has been well summarized in this conference by ZUlch ru) and elsewhere by his co- workers, SchrMer and Wechsler @).

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