By Gabriel M. Danovitch MD

This renowned guide is a pragmatic advisor for physicians, surgeons, nurses, and different pros who deal with kidney transplant sufferers. it's concise, readable, and well-illustrated. Chapters define the foremost matters surrounding renal transplantation and the main winning methods to difficulties bobbing up in temporary and long term sufferer care.

Chapter subject matters comprise immunobiology and immunosuppression, in addition to chapters on surgical procedure, histocompatibility, and the 1st 3 months post-transplant surgical procedure. This completely up to date 5th variation contains new details on strategies for sufferers with end-stage renal ailment, immunosuppressive medicines and protocols for kidney transplantation, and the 1st months following transplant.

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Extra resources for Handbook of Kidney Transplantation

Sample text

Grafts between animals of different inbred strains (allografts) will be rejected. 3. Grafts from a parent to an F1 offspring of two different strains will survive (because the F1 offspring recognizes the graft from the homozygous parent as ‘‘self’’). 4. Grafts from an F1 offspring to a parent will be rejected (because the parent recognizes the graft from the heterozygous F1 as ‘‘nonself’’). These results led to the realization that graft rejection was controlled by genes whose inheritance followed simple mendelian rules.

Kidney Int 2005;67:2089–2100. Meguid El, Nahas A, Bello A. Chronic kidney disease: the global challenge. Lancet 2005; 365:331–340. Mehrotra R, Agarwal R. End-stage renal disease and dialysis. NephSAP 7:374–441;2008 Snyder J, Foley R, Collins A, et al. Prevalence of CKD in the United States: a sensitivity analysis using the National Health and Nutrition Examination Survey (NHANES) 1999–2004. Am J Kidney Dis 2009;53:218–228. Szczech L, Harmon W, Hostetter T, et al. World Kidney Day 2009: Problems and challenges in the emerging epidemic of kidney disease.

Targeting the B7/CD28 pathway holds great promise for achieving antigen-specific unresponsiveness because the timing of transplantation can be controlled so as to coincide with the administration of agents that block CD28 engagement. If blockade of CD28 were only required at the time of initial antigen exposure, a graft recipient would not be exposed to the infectious risks of long-term immunosuppression. In addition, T-cell responses to transplant antigens could be prevented, without the need to identify the specific antigen.

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