By Matthew Menza, Laura Marsh
A pragmatic consultant to the administration of varied medical concerns noticeable in sufferers with Parkinson's illness (PD), this article emphasizes the necessity for coordinated care among many of the pros, in addition to among pros and caregivers. delivering an replace on present advancements within the neurology and administration of PD, in addition to the certainty and remedy of non-motor points of PD, the e-book additionally encompasses a number of subject matters similar to melancholy, psychosis, and anxiousness
Read or Download Psychiatric Issues in Parkinson's Disease: A Practical Guide PDF
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Extra resources for Psychiatric Issues in Parkinson's Disease: A Practical Guide
Sample text
26. Miyasaki JM, Martin W, Suchowersky O et al, Practice parameter: Initiation of treatment for Parkinson’s disease: An evidence-based review: Report of the Quality Standards Subcommittee of the American Academy of Neurology. Neurology 2002; 58:11–17. 27. Wooten GF, Agonists vs levodopa in PD: the thrilla of whitha. Neurology 2003; 60:360–362. 28. Poewe W, Granat R, Geser F, Pharmacologic treatment of Parkinson’s disease. In: Watts RL, Koller WC (eds) Movement Disorders. Neurologic Principles and Practice, 2nd edn.
Neurologic Principles and Practice, 2nd edn. McGraw-Hill: New York, 2004; 233–245. 20. Frucht SJ, Clinical assessments. In: Pahwa R, Lyons KE, Koller WC (eds) Therapy of Parkinson’s Disease, 3rd edn. Marcel Dekker Inc: New York, 2004; 37–52. 21. Hughes AJ, Daniel SE, Kilford L et al, Accuracy of clinical diagnosis of idiopathic Parkinson’s disease: a clinico-pathological study of 100 cases. J Neurol Neurosurg Psychiatry 1992; 55:181–184. 22. Mark MH, Lumping and splitting the Parkinson plus syndromes.
Carbidopa/levodopa 10/100 and 25/250 formulations are also available. The DDI prevents the formation of dopamine peripherally, thereby allowing a lower dose of levodopa to be administered and reducing the risk of associated nausea or vomiting. Levodopa crosses the blood–brain barrier, where it is converted by endogenous aromatic amino acid decarboxylase to dopamine, and then stored in surviving nigrostriatal neuronal terminals. While there is some variation in clinical practice, immediate-release levodopa is usually commenced in a dose of 50–100 mg per day, increasing every 3–4 days until a dose of 50–100 mg three times daily is reached.